Antibodies to varicella-zoster virus modulate antigen distribution but fail to induce viral persistence in vitro.

Varicella-zoster virus (VZV) persists in human sensory ganglia. One of the hypotheses to explain the induction or the maintenance of VZV latency is that it could be promoted by the immune response itself. It is known that in the case of viruses which bud off the infected cell membrane, virus-specifi...

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Bibliografische gegevens

Gepubliceerd in:	J Virol
Hoofdauteurs:	Sadzot-Delvaux, C, Marc, P, Lebon, L, Merville-Louis, M P, Piette, J, Rentier, B
Formaat:	Artigo
Taal:	Inglês
Gepubliceerd in:	American Society for Microbiology (ASM) 1992
Onderwerpen:	Research Article
Online toegang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC240458/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1331534/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.66.12.7499-7504.1992
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