Antibodies to varicella-zoster virus modulate antigen distribution but fail to induce viral persistence in vitro.

Varicella-zoster virus (VZV) persists in human sensory ganglia. One of the hypotheses to explain the induction or the maintenance of VZV latency is that it could be promoted by the immune response itself. It is known that in the case of viruses which bud off the infected cell membrane, virus-specifi...

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Detalles Bibliográficos

Publicado en:	J Virol
Principais autores:	Sadzot-Delvaux, C, Marc, P, Lebon, L, Merville-Louis, M P, Piette, J, Rentier, B
Formato:	Artigo
Idioma:	Inglês
Publicado:	American Society for Microbiology (ASM) 1992
Assuntos:	Research Article
Acceso en liña:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC240458/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1331534/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.66.12.7499-7504.1992
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