Evidence for a novel regulatory pathway for herpes simplex virus gene expression in trigeminal ganglion neurons.

Ítems similars

• Herpes simplex virus thymidine kinase and specific stages of latency in murine trigeminal ganglia.
  per: Jacobson, J G, et al.
  Publicat: (1993)
• Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.
  per: Coen, D M, et al.
  Publicat: (1989)
• Restricted expression of herpes simplex virus lytic genes during establishment of latent infection by thymidine kinase-negative mutant viruses.
  per: Kosz-Vnenchak, M, et al.
  Publicat: (1990)
• Characterization of Neuronal Populations in the Human Trigeminal Ganglion and Their Association with Latent Herpes Simplex Virus-1 Infection
  per: Flowerdew, Sarah E., et al.
  Publicat: (2013)
• A deletion mutant of the latency-associated transcript of herpes simplex virus type 1 reactivates from the latent state with reduced frequency.
  per: Leib, D A, et al.
  Publicat: (1989)