Fusion-defective mutants of mouse hepatitis virus A59 contain a mutation in the spike protein cleavage signal.
Infection of primary mouse glial cell cultures with mouse hepatitis virus strain A59 results in a productive, persistent infection, but without any obvious cytopathic effect. Mutant viruses isolated from infected glial cultures 16 to 18 weeks postinfection replicate with kinetics similar to those of...
Uloženo v:
| Vydáno v: | J Virol |
|---|---|
| Hlavní autoři: | , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Microbiology (ASM)
1993
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC237834/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8392595/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.67.8.4504-4512.1993 |
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