Fusion-defective mutants of mouse hepatitis virus A59 contain a mutation in the spike protein cleavage signal.
Infection of primary mouse glial cell cultures with mouse hepatitis virus strain A59 results in a productive, persistent infection, but without any obvious cytopathic effect. Mutant viruses isolated from infected glial cultures 16 to 18 weeks postinfection replicate with kinetics similar to those of...
Gorde:
| Argitaratua izan da: | J Virol |
|---|---|
| Egile Nagusiak: | , , |
| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
American Society for Microbiology (ASM)
1993
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC237834/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8392595/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.67.8.4504-4512.1993 |
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