Fusion-defective mutants of mouse hepatitis virus A59 contain a mutation in the spike protein cleavage signal.

Infection of primary mouse glial cell cultures with mouse hepatitis virus strain A59 results in a productive, persistent infection, but without any obvious cytopathic effect. Mutant viruses isolated from infected glial cultures 16 to 18 weeks postinfection replicate with kinetics similar to those of...

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Pubblicato in:	J Virol
Autori principali:	Gombold, J L, Hingley, S T, Weiss, S R
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	American Society for Microbiology (ASM) 1993
Soggetti:	Research Article
Accesso online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC237834/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8392595/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.67.8.4504-4512.1993
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