Tissue sparing and functional recovery following experimental traumatic brain injury is provided by treatment with an anti-myelin-associated glycoprotein antibody

Axonal injury is a hallmark of traumatic brain injury (TBI) and is associated with a poor clinical outcome. Following central nervous system injury, axons regenerate poorly, in part due to the presence of molecules associated with myelin that inhibit axonal outgrowth, including myelin-associated gly...

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Main Authors: Thompson, Hilaire J., Marklund, Niklas, LeBold, David G., Morales, Diego M., Keck, Carrie A., Vinson, Mary, Royo, Nicolas C., Grundy, Robert, McIntosh, Tracy K.
Formato: Artigo
Idioma:Inglês
Publicado: 2006
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Article
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC2377452/
https://ncbi.nlm.nih.gov/pubmed/17156367
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1460-9568.2006.05197.x
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