Aldosterone responsiveness of the epithelial sodium channel (ENaC) in colon is increased in a mouse model for Liddle's syndrome

Liddle's syndrome is an autosomal dominant form of human hypertension, caused by gain-of-function mutations of the epithelial sodium channel (ENaC) which is expressed in aldosterone target tissues including the distal colon. We used a mouse model for Liddle's syndrome to investigate ENaC-m...

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Detaylı Bibliyografya
Asıl Yazarlar: Bertog, Marko, Cuffe, John E, Pradervand, Sylvain, Hummler, Edith, Hartner, Andrea, Porst, Markus, Hilgers, Karl F, Rossier, Bernard C, Korbmacher, Christoph
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Blackwell Science Inc 2008
Konular:
Cellular
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2375579/
https://ncbi.nlm.nih.gov/pubmed/18006588
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2007.140459
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