Prion protein attenuates excitotoxicity by inhibiting NMDA receptors

It is well established that misfolded forms of cellular prion protein (PrP [PrP(C)]) are crucial in the genesis and progression of transmissible spongiform encephalitis, whereas the function of native PrP(C) remains incompletely understood. To determine the physiological role of PrP(C), we examine t...

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Enregistré dans:
Détails bibliographiques
Auteurs principaux: Khosravani, Houman, Zhang, Yunfeng, Tsutsui, Shigeki, Hameed, Shahid, Altier, Christophe, Hamid, Jawed, Chen, Lina, Villemaire, Michelle, Ali, Zenobia, Jirik, Frank R., Zamponi, Gerald W.
Format: Artigo
Langue:Inglês
Publié: The Rockefeller University Press 2008
Sujets:
Research Articles
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2364707/
https://ncbi.nlm.nih.gov/pubmed/18443219
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.200711002
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