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TGF-β1 reduces the heterogeneity of astrocytic cyclooxygenase-2 and nitric oxide synthase-2 gene expression in a stimulus-independent manner
Transforming growth factor-β1 (TGF-β1) is upregulated by inflammatory mediators in several neurological diseases/disorders where it either participates in the pathology or provides protection. Often, the biological outcome of TGF-β1 is dependent upon changes in gene expression. Recently, we demonstr...
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| Main Authors: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2007
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2350240/ https://ncbi.nlm.nih.gov/pubmed/18194875 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.prostaglandins.2007.11.004 |
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