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Formyl peptide receptor-mediated ERK1/2 activation occurs through G(i) and is not dependent on β-arrestin1/2
Formyl peptide receptor (FPR) and C5a receptor (C5aR) are chemoattractant G protein-coupled receptors (GPCRs) involved in the innate immune response against bacterial infections and tissue injury. Like other GPCRs, they recruit β-arrestin1/2 to the plasma membrane and activate the extracellular sign...
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| Autors principals: | , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2007
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2330276/ https://ncbi.nlm.nih.gov/pubmed/18060741 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cellsig.2007.11.002 |
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