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Formyl peptide receptor-mediated ERK1/2 activation occurs through G(i) and is not dependent on β-arrestin1/2

Formyl peptide receptor (FPR) and C5a receptor (C5aR) are chemoattractant G protein-coupled receptors (GPCRs) involved in the innate immune response against bacterial infections and tissue injury. Like other GPCRs, they recruit β-arrestin1/2 to the plasma membrane and activate the extracellular sign...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
मुख्य लेखकों: Gripentrog, Jeannie M., Miettinen, Heini M.
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: 2007
विषय:
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC2330276/
https://ncbi.nlm.nih.gov/pubmed/18060741
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cellsig.2007.11.002
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