The sensitivity of Cockayne's syndrome cells to DNA-damaging agents is not due to defective transcription-coupled repair of active genes.

Two of the hallmarks of Cockayne's syndrome (CS) are the hypersensitivity of cells to UV light and the lack of recovery of the ability to synthesize RNA following exposure of cells to UV light, in spite of the normal repair capacity at the overall genome level. The prolonged repressed RNA synthesis...

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Detalhes bibliográficos

Publicado no:	Mol Cell Biol
Principais autores:	van Oosterwijk, M F, Versteeg, A, Filon, R, van Zeeland, A A, Mullenders, L H
Formato:	Artigo
Idioma:	Inglês
Publicado em:	Taylor & Francis 1996
Assuntos:	Research Article
Acesso em linha:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC231442/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8754844/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.16.8.4436
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