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Both coding exons of the c-myc gene contribute to its posttranscriptional regulation in the quiescent liver and regenerating liver and after protein synthesis inhibition.

In vivo, the steady-state level of c-myc mRNA is mainly controlled by posttranscriptional mechanisms. Using a panel of transgenic mice in which various versions of the human c-myc proto-oncogene were under the control of major histocompatibility complex H-2Kb class I regulatory sequences, we have sh...

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Detalhes bibliográficos
Publicado no:Mol Cell Biol
Principais autores: Lavenu, A, Pistoi, S, Pournin, S, Babinet, C, Morello, D
Formato: Artigo
Idioma:Inglês
Publicado em: Taylor & Francis 1995
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC230681/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7623834/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.15.8.4410
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