Akt-Directed Glucose Metabolism Can Prevent Bax Conformation Change and Promote Growth Factor-Independent Survival

The serine/threonine kinase Akt is a component of many receptor signal transduction pathways and can prevent cell death following growth factor withdrawal. Here, we show that Akt inhibition of cell death is not dependent on new protein translation. Instead, Akt inhibition of cell death requires gluc...

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שמור ב:
מידע ביבליוגרפי
Main Authors: Rathmell, Jeffrey C., Fox, Casey J., Plas, David R., Hammerman, Peter S., Cinalli, Ryan M., Thompson, Craig B.
פורמט: Artigo
שפה:Inglês
יצא לאור: American Society for Microbiology 2003
נושאים:
Cell Growth and Development
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC230333/
https://ncbi.nlm.nih.gov/pubmed/14517300
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.23.20.7315-7328.2003
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