ACh-induced endothelial NO synthase translocation, NO release and vasodilatation in the hamster microcirculation in vivo

Studies in cultured cells show that activation of endothelial nitric oxide (NO) synthase (eNOS) requires the dissociation of this enzyme from its inhibitory association with caveolin-1 (Cav-1), and perhaps its translocation from plasma membrane caveolae to other cellular compartments. We investigate...

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Autors principals: Figueroa, Xavier F, González, Daniel R, Martínez, Agustín D, Durán, Walter N, Boric, Mauricio P
Format: Artigo
Idioma:Inglês
Publicat: Blackwell Science Inc 2002
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Original Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2290640/
https://ncbi.nlm.nih.gov/pubmed/12411531
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2002.021972
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