The Gammaherpesvirus 68 Latency-Associated Nuclear Antigen Homolog Is Critical for the Establishment of Splenic Latency

Lignende værker

• The Murine Gammaherpesvirus 68 v-Cyclin Is a Critical Regulator of Reactivation from Latency
  af: van Dyk, Linda F., et al.
  Udgivet: (2000)
• Disruption of the M2 Gene of Murine Gammaherpesvirus 68 Alters Splenic Latency following Intranasal, but Not Intraperitoneal, Inoculation
  af: Jacoby, Meagan A., et al.
  Udgivet: (2002)
• B Cells Regulate Murine Gammaherpesvirus 68 Latency
  af: Weck, Karen E., et al.
  Udgivet: (1999)
• Establishment and Maintenance of Long-Term Murine Gammaherpesvirus 68 Latency in B Cells in the Absence of CD40
  af: Willer, David O., et al.
  Udgivet: (2005)
• Long-Term Latent Murine Gammaherpesvirus 68 Infection Is Preferentially Found within the Surface Immunoglobulin D-Negative Subset of Splenic B Cells In Vivo
  af: Willer, David O., et al.
  Udgivet: (2003)