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Detecting hidden sequence propensity for amyloid fibril formation

The preponderance of evidence implicates protein misfolding in many unrelated human diseases. In all cases, normal correctly folded proteins transform from their proper native structure into an abnormal β-rich structure known as amyloid fibril. Here we introduce a computational algorithm to detect n...

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Detalles Bibliográficos
Main Authors: Yoon, Sukjoon, Welsh, William J.
Formato: Artigo
Idioma:Inglês
Publicado: Cold Spring Harbor Laboratory Press 2004
Assuntos:
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC2279810/
https://ncbi.nlm.nih.gov/pubmed/15273309
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1110/ps.04790604
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