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Sepsis-induced organ failure is mediated by different pathways in the kidney and liver: acute renal failure is dependent on MyD88 but not renal cell apoptosis
BACKGROUND: Toll-like receptors (TLRs) are important in the pathophysiology of sepsis. Myeloid differentiation factor 88 (MyD88) is a key molecule involved in signal transduction by multiple TLRs. The objective of this study was to investigate the contribution of TLR4 and MyD88 in acute renal failur...
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| Main Authors: | , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2006
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2271059/ https://ncbi.nlm.nih.gov/pubmed/16518342 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.ki.5000165 |
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