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In vivo alpha-synuclein overexpression in rodents: a useful model of Parkinson’s disease?
Mutations in alpha-synuclein were the first genetic defect linked to Parkinson’s disease (PD). The relevance of alpha-synuclein to sporadic PD is strongly supported by the presence of alpha-synuclein aggregates in neurons of patients. This has prompted the development of numerous animal models based...
Αποθηκεύτηκε σε:
| Κύριος συγγραφέας: | |
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| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2007
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2262051/ https://ncbi.nlm.nih.gov/pubmed/17949715 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2007.08.006 |
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