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Toll-Like Receptor 5-Deficient Mice Have Dysregulated Intestinal Gene Expression and Nonspecific Resistance to Salmonella-Induced Typhoid-Like Disease

The recognition of flagellin by Toll-like receptor 5 (TLR5) is the dominant means by which model intestinal epithelia activate proinflammatory gene expression in response to Salmonella enterica. The role of the flagellin-TLR5 interaction in vivo has been addressed primarily via studies that use flag...

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מידע ביבליוגרפי
Main Authors: Vijay-Kumar, Matam, Aitken, Jesse D., Kumar, Amrita, Neish, Andrew S., Uematsu, Satoshi, Akira, Shizuo, Gewirtz, Andrew T.
פורמט: Artigo
שפה:Inglês
יצא לאור: American Society for Microbiology (ASM) 2008
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2258833/
https://ncbi.nlm.nih.gov/pubmed/18195036
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/IAI.01491-07
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