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MIP-2 causes differential activation of RhoA in mouse aortic versus pulmonary artery endothelial cells
Previously, we have shown that endothelial cell chemotaxis to the proangiogenic chemokine MIP-2 (macrophage inflammatory protein-2), is much greater in mouse aortic endothelial cells (EC) than pulmonary arterial endothelial cells (PA EC). This was true despite the observation that both cell types di...
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| Main Authors: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2007
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2258091/ https://ncbi.nlm.nih.gov/pubmed/17662312 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.mvr.2007.06.007 |
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