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Absence of dystrophin in mice reduces NO-dependent vascular function and vascular density: total recovery after a treatment with the aminoglycoside gentamicin
Mutations in the dystrophin gene causing Duchenne’s muscular dystrophy (DMD), lead to pre-mature stop codons. In mdx mice, a model for DMD, they can be suppressed by aminoglycosides such as gentamicin. Dystrophin is likely to play a role in flow (shear stress) mediated endothelium-dependent dilation...
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| Asıl Yazarlar: | , , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Lippincott Williams & Wilkins
2004
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2233851/ https://ncbi.nlm.nih.gov/pubmed/14751810 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/01.ATV.0000118683.99628.42 |
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