Cl(−) transport by cystic fibrosis transmembrane conductance regulator (CFTR) contributes to the inhibition of epithelial Na(+) channels (ENaCs) in Xenopus oocytes co-expressing CFTR and ENaC

1. Epithelial Na(+) channels (ENaCs) are inhibited by the cystic fibrosis transmembrane conductance regulator (CFTR) when CFTR is activated by protein kinase A. Since cAMP-dependent activation of CFTR Cl(−) conductance is defective in cystic fibrosis (CF), ENaC currents are not inhibited by CFTR. Th...

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Bibliografiska uppgifter
Huvudupphovsmän: Briel, M, Greger, R, Kunzelmann, K
Materialtyp: Artigo
Språk:Inglês
Publicerad: Blackwell Science Inc 1998
Ämnen:
Original Articles
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2230925/
https://ncbi.nlm.nih.gov/pubmed/9518736
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7793.1998.825bp.x
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