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Cl(−) transport by cystic fibrosis transmembrane conductance regulator (CFTR) contributes to the inhibition of epithelial Na(+) channels (ENaCs) in Xenopus oocytes co-expressing CFTR and ENaC
1. Epithelial Na(+) channels (ENaCs) are inhibited by the cystic fibrosis transmembrane conductance regulator (CFTR) when CFTR is activated by protein kinase A. Since cAMP-dependent activation of CFTR Cl(−) conductance is defective in cystic fibrosis (CF), ENaC currents are not inhibited by CFTR. Th...
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| Asıl Yazarlar: | , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Blackwell Science Inc
1998
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2230925/ https://ncbi.nlm.nih.gov/pubmed/9518736 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7793.1998.825bp.x |
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