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β(2)-microglobulin–deficient Mice Are Resistant to Bullous Pemphigoid

Recent understanding of the mechanism of immunoglobulin G (IgG) catabolism has yielded new insight into antibody-mediated diseases. We proposed that β(2)-microglobulin (β(2)m)–deficient mice have been protected from systemic lupus erythematosis (SLE)–like syndromes because they lack the β(2)m-associ...

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Detalhes bibliográficos
Main Authors: Liu, Zhi, Roopenian, Derry C., Zhou, Xiaoye, Christianson, Greg J., Diaz, Luis A., Sedmak, Daniel D., Anderson, Clark L.
Formato: Artigo
Idioma:Inglês
Publicado em: The Rockefeller University Press 1997
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2199010/
https://ncbi.nlm.nih.gov/pubmed/9271593
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