טוען...
Growth Inhibition and Apoptosis Due to Restoration of E2A Activity in T Cell Acute Lymphoblastic Leukemia Cells
Two models have been proposed for the molecular mechanism by which the Tal1 oncogene causes T cell acute lymphoblastic leukemia (T-ALL). The activation model suggests that Tal1 as heterodimers with the E2A transcription factor activates the expression of oncogenes. The inhibition model postulates th...
שמור ב:
Main Authors: | , , |
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פורמט: | Artigo |
שפה: | Inglês |
יצא לאור: |
The Rockefeller University Press
1999
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נושאים: | |
גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2192921/ https://ncbi.nlm.nih.gov/pubmed/9927512 |
תגים: |
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