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Lymphocyte dysfunction after DNA damage by toxic oxygen species. A model of immunodeficiency

The metabolic causes for immune impairment in patients with severe chronic inflammatory diseases have not been clearly defined. Recently, the overproduction of poly(ADP-ribose) in resting lymphocytes with unrepaired DNA strand breaks has been suggested to contribute to immune dysfunction in adenosin...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: The Rockefeller University Press 1986
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2188042/
https://ncbi.nlm.nih.gov/pubmed/3950545
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