δPKC Participates in the Endoplasmic Reticulum Stress-Induced Response in Cultured Cardiac Myocytes and Ischemic Heart

The cellular response to excessive endoplasmic reticulum (ER) stress includes the activation of signaling pathways, which lead to apoptotic cell death. Here we show that treatment of cultured cardiac myocytes with tunicamycin, an agent that induces ER stress, causes the rapid translocation of δPKC t...

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Hlavní autoři: Qi, Xin, Vallentin, Alice, Churchill, Eric, Mochly-Rosen, Daria
Médium: Artigo
Jazyk:Inglês
Vydáno: 2007
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2185772/
https://ncbi.nlm.nih.gov/pubmed/17825316
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2007.07.061
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