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Pathogenic point mutations in a transmembrane domain of the ε subunit increase the Ca(2+) permeability of the human endplate ACh receptor
The ε subunit of the human endplate ACh receptor (AChR) is a key determinant of the large fraction of the ACh-evoked current carried by Ca(2+) ions (P(f)). Consequently, missense mutations in the ε subunit are potential targets for altering the P(f) of human AChR. In this paper we investigate the ef...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Blackwell Science Inc
2007
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2151372/ https://ncbi.nlm.nih.gov/pubmed/17272341 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2007.127977 |
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