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The human adult subtype ACh receptor channel has high Ca(2+) permeability and predisposes to endplate Ca(2+) overloading
Slow-channel congenital myasthenic syndrome, caused by mutations in subunits of the endplate ACh receptor (AChR), results in prolonged synaptic currents and excitotoxic injury of the postsynaptic region by Ca(2+) overloading. The Ca(2+) overloading could be due entirely to the prolonged openings of...
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| Main Authors: | , , , , |
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| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
Blackwell Science Inc
2006
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1779694/ https://ncbi.nlm.nih.gov/pubmed/16527851 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2006.108092 |
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