Mutations that create new promoters suppress the sigma 54 dependence of glnA transcription in Escherichia coli.

Escherichia coli rpoN mutants lack sigma 54 and are therefore unable to initiate the transcription of glnA at glnAp2, which is required for the production of a high intracellular concentration of glutamine synthetase. We have found that the dependence on sigma 54 can be overcome by mutations that ha...

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Detaylı Bibliyografya
Asıl Yazarlar: Reitzer, L J, Bueno, R, Cheng, W D, Abrams, S A, Rothstein, D M, Hunt, T P, Tyler, B, Magasanik, B
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 1987
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC213741/
https://ncbi.nlm.nih.gov/pubmed/2887548
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