Mutations that create new promoters suppress the sigma 54 dependence of glnA transcription in Escherichia coli.
Escherichia coli rpoN mutants lack sigma 54 and are therefore unable to initiate the transcription of glnA at glnAp2, which is required for the production of a high intracellular concentration of glutamine synthetase. We have found that the dependence on sigma 54 can be overcome by mutations that ha...
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| 出版年: | J Bacteriol |
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| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology (ASM)
1987
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC213741/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2887548/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jb.169.9.4279-4284.1987 |
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