Escherichia coli strains with multiple DNA repair defects are hyperinduced for the SOS response.
Escherichia coli strains defective for the repair of apurinic/apyrimidinic sites and for the UvrABC excision repair pathway could be constructed if they also carried a mutation in ung, which encodes uracil glycosylase, or sulA, which encodes an SOS-inducible inhibitor of septation. The resultant str...
Uloženo v:
| Vydáno v: | J Bacteriol |
|---|---|
| Hlavní autor: | |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Microbiology (ASM)
1990
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC213313/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2115878/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jb.172.8.4719-4720.1990 |
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