Escherichia coli strains with multiple DNA repair defects are hyperinduced for the SOS response.
Escherichia coli strains defective for the repair of apurinic/apyrimidinic sites and for the UvrABC excision repair pathway could be constructed if they also carried a mutation in ung, which encodes uracil glycosylase, or sulA, which encodes an SOS-inducible inhibitor of septation. The resultant str...
Sparad:
| I publikationen: | J Bacteriol |
|---|---|
| Huvudupphov: | |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Utgiven: |
American Society for Microbiology (ASM)
1990
|
| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC213313/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2115878/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jb.172.8.4719-4720.1990 |
| Taggar: |
Inga taggar, Lägg till första taggen!
|