Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer

Nitrite (NO(2) (−)) is an intrinsic signaling molecule that is reduced to NO during ischemia and limits apoptosis and cytotoxicity at reperfusion in the mammalian heart, liver, and brain. Although the mechanism of nitrite-mediated cytoprotection is unknown, NO is a mediator of the ischemic precondit...

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Hlavní autoři: Shiva, Sruti, Sack, Michael N., Greer, James J., Duranski, Mark, Ringwood, Lorna A., Burwell, Lindsay, Wang, Xunde, MacArthur, Peter H., Shoja, Amir, Raghavachari, Nalini, Calvert, John W., Brookes, Paul S., Lefer, David J., Gladwin, Mark T.
Médium: Artigo
Jazyk:Inglês
Vydáno: The Rockefeller University Press 2007
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2118713/
https://ncbi.nlm.nih.gov/pubmed/17682069
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20070198
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