Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer

Nitrite (NO(2) (−)) is an intrinsic signaling molecule that is reduced to NO during ischemia and limits apoptosis and cytotoxicity at reperfusion in the mammalian heart, liver, and brain. Although the mechanism of nitrite-mediated cytoprotection is unknown, NO is a mediator of the ischemic precondit...

Полное описание

Сохранить в:
Библиографические подробности
Главные авторы: Shiva, Sruti, Sack, Michael N., Greer, James J., Duranski, Mark, Ringwood, Lorna A., Burwell, Lindsay, Wang, Xunde, MacArthur, Peter H., Shoja, Amir, Raghavachari, Nalini, Calvert, John W., Brookes, Paul S., Lefer, David J., Gladwin, Mark T.
Формат: Artigo
Язык:Inglês
Опубликовано: The Rockefeller University Press 2007
Предметы:
Articles
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC2118713/
https://ncbi.nlm.nih.gov/pubmed/17682069
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20070198
Метки: Добавить метку
Нет меток, Требуется 1-ая метка записи!

Схожие документы