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K(ATP) channel deficiency in mouse flexor digitorum brevis causes fibre damage and impairs Ca(2+) release and force development during fatigue in vitro
Activation of the K(ATP) channels results in faster fatigue rates as the channels depress action potential amplitude, whereas abolishing the channel activity has no effect in whole extensor digitorum longus (EDL) and soleus muscles. In this study, we examined the effects of abolished K(ATP) channel...
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| Main Authors: | , , , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Blackwell Science Inc
2007
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2075337/ https://ncbi.nlm.nih.gov/pubmed/17510189 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2007.130955 |
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