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K(ATP) channel deficiency in mouse flexor digitorum brevis causes fibre damage and impairs Ca(2+) release and force development during fatigue in vitro

Activation of the K(ATP) channels results in faster fatigue rates as the channels depress action potential amplitude, whereas abolishing the channel activity has no effect in whole extensor digitorum longus (EDL) and soleus muscles. In this study, we examined the effects of abolished K(ATP) channel...

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Detalhes bibliográficos
Main Authors: Cifelli, Carlo, Bourassa, François, Gariépy, Louise, Banas, Krystyna, Benkhalti, Maria, Renaud, Jean-Marc
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Science Inc 2007
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2075337/
https://ncbi.nlm.nih.gov/pubmed/17510189
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2007.130955
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