β-Liddle mutation of the epithelial sodium channel increases alveolar fluid clearance and reduces the severity of hydrostatic pulmonary oedema in mice

Lignende værker

• ENaC-mediated alveolar fluid clearance and lung fluid balance depend on the channel-activating protease 1
  af: Planès, Carole, et al.
  Udgivet: (2010)
• VEGF levels in the alveolar compartment do not distinguish between ARDS and hydrostatic pulmonary oedema
  af: Ware, L.B., et al.
  Udgivet: (2005)
• Conditioned media from mesenchymal stromal cells restore sodium transport and preserve epithelial permeability in an in vitro model of acute alveolar injury
  af: Goolaerts, Arnaud, et al.
  Udgivet: (2014)
• Vasopressin-stimulated CFTR Cl(−) currents are increased in the renal collecting duct cells of a mouse model of Liddle's syndrome
  af: Chang, Chiz-Tzung, et al.
  Udgivet: (2005)
• Synergistic Activation of ENaC by Three Membrane-bound Channel-activating Serine Proteases (mCAP1, mCAP2, and mCAP3) and Serum- and Glucocorticoid-regulated Kinase (Sgk1) in Xenopus Oocytes
  af: Vuagniaux, Grégoire, et al.
  Udgivet: (2002)