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Up-regulation of the inward rectifier K(+) current (I(K1)) in the mouse heart accelerates and stabilizes rotors
Previous studies have suggested an important role for the inward rectifier K(+) current (I(K1)) in stabilizing rotors responsible for ventricular tachycardia (VT) and fibrillation (VF). To test this hypothesis, we used a line of transgenic mice (TG) overexpressing Kir 2.1–green fluorescent protein (...
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| Huvudupphovsmän: | , , , , , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
Blackwell Science Inc
2007
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2075137/ https://ncbi.nlm.nih.gov/pubmed/17095564 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2006.121475 |
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