Up-regulation of the inward rectifier K(+) current (I(K1)) in the mouse heart accelerates and stabilizes rotors

Previous studies have suggested an important role for the inward rectifier K(+) current (I(K1)) in stabilizing rotors responsible for ventricular tachycardia (VT) and fibrillation (VF). To test this hypothesis, we used a line of transgenic mice (TG) overexpressing Kir 2.1–green fluorescent protein (...

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Bibliografiska uppgifter
Huvudupphovsmän: Noujaim, Sami F, Pandit, Sandeep V, Berenfeld, Omer, Vikstrom, Karen, Cerrone, Marina, Mironov, Sergey, Zugermayr, Michelle, Lopatin, Anatoli N, Jalife, José
Materialtyp: Artigo
Språk:Inglês
Publicerad: Blackwell Science Inc 2007
Ämnen:
Cardiovascular
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2075137/
https://ncbi.nlm.nih.gov/pubmed/17095564
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2006.121475
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