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β4 integrin activates a Shp2–Src signaling pathway that sustains HGF-induced anchorage-independent growth
Despite being a cell–matrix adhesion molecule, β4 integrin can prompt the multiplication of neoplastic cells dislodged from their substrates (anchorage-independent growth). However, the molecular events underlying this atypical behavior remain partly unexplored. We found that activation of the Met r...
Tallennettuna:
| Päätekijät: | , , |
|---|---|
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
The Rockefeller University Press
2006
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2064708/ https://ncbi.nlm.nih.gov/pubmed/17158954 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.200605114 |
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