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Lack of MK2 Inhibits Myofibroblast Formation and Exacerbates Pulmonary Fibrosis
Fibroblasts play a major role in tissue repair and remodeling. Their differentiation into myofibroblasts, marked by increased expression of smooth muscle–specific α-actin (α-SMA), is believed to be important in wound healing and fibrosis. We have recently described a role for MK2 in this phenotypic...
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| Hlavní autoři: | , , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Thoracic Society
2007
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2048679/ https://ncbi.nlm.nih.gov/pubmed/17600313 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1165/rcmb.2007-0077OC |
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