Cellular and Viral Factors Regulate the Varicella-Zoster Virus gE Promoter during Viral Replication

Samankaltaisia teoksia

• Mutagenesis of Varicella-Zoster Virus Glycoprotein B: Putative Fusion Loop Residues Are Essential for Viral Replication, and the Furin Cleavage Motif Contributes to Pathogenesis in Skin Tissue In Vivo
  Tekijä: Oliver, Stefan L., et al.
  Julkaistu: (2009)
• Regulation of the ORF61 Promoter and ORF61 Functions in Varicella-Zoster Virus Replication and Pathogenesis
  Tekijä: Wang, Li, et al.
  Julkaistu: (2009)
• Functions of Varicella-Zoster Virus ORF23 Capsid Protein in Viral Replication and the Pathogenesis of Skin Infection
  Tekijä: Chaudhuri, Vaishali, et al.
  Julkaistu: (2008)
• Deletion of the First Cysteine-Rich Region of the Varicella-Zoster Virus Glycoprotein E Ectodomain Abolishes the gE and gI Interaction and Differentially Affects Cell-Cell Spread and Viral Entry
  Tekijä: Berarducci, Barbara, et al.
  Julkaistu: (2009)
• Identification of a Hydrophobic Domain in Varicella-Zoster Virus ORF61 Necessary for ORF61 Self-Interaction, Viral Replication, and Skin Pathogenesis
  Tekijä: Wang, Li, et al.
  Julkaistu: (2013)