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JunB suppresses cell proliferation by transcriptional activation of p16(INK4a) expression

A role for the transcription factor JunB in proliferation control was investigated in genetically modified mouse fibroblasts. Increased JunB expression induced high levels of the cyclin-dependent kinase inhibitor p16(INK4a), leading to premature senescence in primary cells and reduced proliferation...

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Autori principali: Passegué, Emmanuelle, Wagner, Erwin F.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Oxford University Press 2000
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC203376/
https://ncbi.nlm.nih.gov/pubmed/10856241
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/19.12.2969
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