Animal model of human disease. Crigler-Najjar Syndrome. Animal model: hereditary nonhemolytic unconjugated hyperbilirubinemia in Gunn rats.

Ítems similars

• Successful Plasmapheresis for Acute and Severe Unconjugated Hyperbilirubinemia in a Child with Crigler Najjar Type I Syndrome
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• Animal model of human disease. Increased sensitivity to polychlorinated biphenyls. Animal model: gunn rats with hereditary hyperbilirubinemia.
  per: Calabrese, E. J.
  Publicat: (1978)
• Gilbert or Crigler–Najjar syndrome? Neonatal severe unconjugated hyperbilirubinemia with P364L UGT1A1 homozygosity
  per: Laura Cozzi, et al.
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• Severe neonatal hyperbilirubinemia in Crigler‐Najjar syndrome model mice can be reversed with zinc protoporphyrin
  per: Ryoichi Fujiwara, et al.
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• Severe neonatal hyperbilirubinemia in Crigler‐Najjar syndrome model mice can be reversed with zinc protoporphyrin
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