Animal model of human disease. Crigler-Najjar Syndrome. Animal model: hereditary nonhemolytic unconjugated hyperbilirubinemia in Gunn rats.

Podobne knjige/članki

• Successful Plasmapheresis for Acute and Severe Unconjugated Hyperbilirubinemia in a Child with Crigler Najjar Type I Syndrome
  od: Sellier, Anne Laure, et al.
  Izdano: (2011)
• Animal model of human disease. Increased sensitivity to polychlorinated biphenyls. Animal model: gunn rats with hereditary hyperbilirubinemia.
  od: Calabrese, E. J.
  Izdano: (1978)
• Gilbert or Crigler–Najjar syndrome? Neonatal severe unconjugated hyperbilirubinemia with P364L UGT1A1 homozygosity
  od: Laura Cozzi, et al.
  Izdano: (2022-04-01)
• Severe neonatal hyperbilirubinemia in Crigler‐Najjar syndrome model mice can be reversed with zinc protoporphyrin
  od: Ryoichi Fujiwara, et al.
  Izdano: (2017-10-01)
• Severe neonatal hyperbilirubinemia in Crigler‐Najjar syndrome model mice can be reversed with zinc protoporphyrin
  od: Fujiwara, Ryoichi, et al.
  Izdano: (2017)