Myocardial ischemia results in tetrahydrobiopterin (BH(4)) oxidation with impaired endothelial function ameliorated by BH(4)

Coronary vasodilation is impaired in the postischemic heart with a loss of endothelial nitric oxide synthase (eNOS) activity, but the mechanisms underlying ischemia-induced eNOS dysfunction are not understood. For nitric oxide (NO) synthesis, eNOS requires the redox-sensitive cofactor tetrahydrobiop...

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Bibliografske podrobnosti
Main Authors: Dumitrescu, Cristian, Biondi, Roberto, Xia, Yong, Cardounel, Arturo J., Druhan, Lawrence J., Ambrosio, Giuseppe, Zweier, Jay L.
Format: Artigo
Jezik:Inglês
Izdano: National Academy of Sciences 2007
Teme:
Biological Sciences
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC1986616/
https://ncbi.nlm.nih.gov/pubmed/17848522
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0702986104
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