Naturally occurring and bioengineered apoA-I mutations that inhibit the conversion of discoidal to spherical HDL: the abnormal HDL phenotypes can be corrected by treatment with LCAT

Antzeko izenburuak

• The Carboxy-Terminal Region of apoA-I is Required for the ABCA1-Dependent Formation of α-HDL but not preβ-HDL Particles In Vivo
  nork: Chroni, Angeliki, et al.
  Argitaratua: (2007)
• ApoA-IV promotes the biogenesis of apoA-IV-containing HDL particles with the participation of ABCA1 and LCAT
  nork: Duka, Adelina, et al.
  Argitaratua: (2013)
• Alteration of negatively charged residues in the 89 to 99 domain of apoA-I affects lipid homeostasis and maturation of HDL
  nork: Kateifides, Andreas K., et al.
  Argitaratua: (2011)
• Effect of apoA-I Mutations in the Capacity of Reconstituted HDL to Promote ABCG1-Mediated Cholesterol Efflux
  nork: Daniil, Georgios, et al.
  Argitaratua: (2013)
• Significance of the hydrophobic residues 225–230 of apoA-I for the biogenesis of HDL(1)
  nork: Fotakis, Panagiotis, et al.
  Argitaratua: (2013)