Modulation of microglial/macrophage activation by macrophage inhibitory factor (TKP) or tuftsin (TKPR) attenuates the disease course of experimental autoimmune encephalomyelitis

BACKGROUND: Myelin Oligodendrocyte Glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE) is the most commonly used mouse model for multiple sclerosis (MS). During the of progression of EAE, microglia, the immunocompetent cells of the brain, become activated and accumulate around...

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Bibliografiske detaljer
Main Authors: Bhasin, Madhuri, Wu, Muzhou, Tsirka, Stella E
Format: Artigo
Sprog:Inglês
Udgivet: BioMed Central 2007
Fag:
Research Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1937009/
https://ncbi.nlm.nih.gov/pubmed/17634104
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2172-8-10
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