An ultrasensitive Ca(2+)/calmodulin-dependent protein kinase II-protein phosphatase 1 switch facilitates specificity in postsynaptic calcium signaling
The strength of hippocampal synapses can be persistently increased by signals that activate Ca(2+)/calmodulin-dependent protein kinase II (CaMKII). This CaMKII-dependent long-term potentiation is important for hippocampal learning and memory. In this work we show that CaMKII exhibits an intriguing s...
Tallennettuna:
| Julkaisussa: | Proc Natl Acad Sci U S A |
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| Päätekijät: | , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
National Academy of Sciences
2003
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC193592/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12928489/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.1932759100 |
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