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Increased lymphomagenicity and restored disease specificity of AML1 site (core) mutant SL3-3 murine leukemia virus by a second-site enhancer variant evolved in vivo.

SL3-3 is a highly T-lymphomagenic murine retrovirus. The major genetic determinant of disease is the transcriptional enhancer, which consists of a repeated region with densely packed binding sites for several transcription factors, including AML1 (also known as core binding factor and polyoma enhanc...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Ethelberg, S, Lovmand, J, Schmidt, J, Luz, A, Pedersen, F S
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 1997
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC192069/
https://ncbi.nlm.nih.gov/pubmed/9311802
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